About Garret Yount

Garret Yount

Garret Yount, PhD, is a Scientist at the Institute of Noetic Sciences and the Director of the Mind-Body Medicine Research Group at California Pacific Medical Center Research Institute. Dr. Yount’s primary interest is bridging molecular neurobiology with aspects of consciousness and the nonmaterial mind. He began his formal studies in consciousness research with a small grant from the Institute of Noetic Sciences in 1995, which allowed him to work with biofield practitioners and spiritual healers in the laboratory.

Dr. Yount studied molecular and cellular biology at Pennsylvania State University, before completing a Ph.D. in Neurobiology and Behavior at the State University of New York at Stony Brook. During his postdoctoral training at the University of California, San Francisco, he was awarded a fellowship from the Robert Steel Foundation for Pediatric Cancer Research.

Dr Yount has established a track record in obtaining research funding from both federal and private agencies, and has had the opportunity to conduct carefully controlled experiments with biofield practitioners and spiritual healers from the United States, Canada, China, Japan, India, Russia, Hungary, Brazil, and Sri Lanka. Through collaboration with the Institute for Health and Healing at California Pacific Medical Center, Dr. Yount is also studying physiologic changes associated with stress reduction and positive emotions, and how these changes are affected by novel interventions including Equine Assisted Therapies and Expressive Arts Therapy. Results of a pilot study of the effects of a novel Expressive Arts Therapy created at CPMC for hospitalized children showed reduced levels of the stress hormone cortisol and were published in the journal Pediatric Reports.

See also Garret's publications in the online IONS Library

Expressive Arts Therapy with Hospitalized Children: A Pilot Study of Co-Creating Healing Sock Creatures(©).
Siegel J, Iida H, Rachlin K, Yount G.
Journal of pediatric nursing. 2016; 31(1):92-8.

Impact of prospectively determined A118G polymorphism on treatment response to injectable naltrexone among methamphetamine-dependent patients: an open-label, pilot study.
Pal R, Mendelson JE, Flower K, Garrison K, Yount G, et al.
Journal of addiction medicine. 2015; 9(2):130-5. NIHMSID: NIHMS649984

HCMV glycoprotein B is expressed in primary glioblastomas and enhances growth and invasiveness via PDGFR-alpha activation.
Cobbs C, Khan S, Matlaf L, McAllister S, Zider A, et al.
Oncotarget. 2014; 5(4):1091-100.

Infrasound sensitizes human glioblastoma cells to cisplatin-induced apoptosis.
Rachlin K, Moore DH, Yount G.
Integrative cancer therapies. 2013; 12(6):517-27.

Gene expression analysis in combat veterans with and without post‑traumatic stress disorder.
Hollifield M, Moore D, Yount G.
Molecular medicine reports. 2013; 8(1):238-44.

Expressive arts therapy for hospitalized children: a pilot study measuring cortisol levels.
Yount G, Rachlin K, Siegel J.
Pediatric reports. 2013; 5(2):28-30.

Id-1 is a key transcriptional regulator of glioblastoma aggressiveness and a novel therapeutic target.
Soroceanu L, Murase R, Limbad C, Singer E, Allison J, et al.
Cancer research. 2013; 73(5):1559-69. NIHMSID: NIHMS429099

Evaluation of biofield treatment dose and distance in a model of cancer cell death.
Yount G, Patil S, Dave U, Alves-dos-Santos L, Gon K, et al.
Journal of alternative and complementary medicine (New York, N.Y.). 2013; 19(2):124-7.

Biofield research: a roundtable discussion of scientific and methodological issues.
Hammerschlag R, Jain S, Baldwin AL, Gronowicz G, Lutgendorf SK, et al.
Journal of alternative and complementary medicine (New York, N.Y.). 2012; 18(12):1081-6.

The effect of emotional freedom techniques on stress biochemistry: a randomized controlled trial.
Church D, Yount G, Brooks AJ.
The Journal of nervous and mental disease. 2012; 200(10):891-6.

Vascular endothelial platelet endothelial cell adhesion molecule 1 (PECAM-1) regulates advanced metastatic progression.
DeLisser H, Liu Y, Desprez PY, Thor A, Briasouli P, et al.
Proceedings of the National Academy of Sciences of the United States of America. 2010; 107(43):18616-21.

Biochemical mechanism of acetaminophen (APAP) induced toxicity in melanoma cell lines.
Vad NM, Yount G, Moore D, Weidanz J, Moridani MY.
Journal of pharmaceutical sciences. 2009; 98(4):1409-25. NIHMSID: NIHMS64733

Biochemical mechanism of acetylsalicylic acid (Aspirin) selective toxicity toward melanoma cell lines.
Vad NM, Yount G, Moridani MY.
Melanoma research. 2008; 18(6):386-99.

A novel camptothecin analog with enhanced antitumor activity.
Yount G, Yang Y, Wong B, Wang HJ, Yang LX.
Anticancer research. 2007; 27(5A):3173-8.

Evaluating biofield treatments in a cell culture model of oxidative stress.
Mager J, Moore D, Bendl D, Wong B, Rachlin K, et al.
Explore (New York, N.Y.). 2007; 3(4):386-90.

Radiation response of cultured human cells is unaffected by Johrei.
Hall Z, Luu T, Moore D, Yount G.
Evidence-based complementary and alternative medicine : eCAM. 2007; 4(2):191-4.

Independent motile microplast formation correlates with glioma cell invasiveness.
Yount G, Taft RJ, Luu T, Rachlin K, Moore D, et al.
Journal of neuro-oncology. 2007; 81(2):113-21.

Radiation-induced caspase-8 mediates p53-independent apoptosis in glioma cells.
Afshar G, Jelluma N, Yang X, Basila D, Arvold ND, et al.
Cancer research. 2006; 66(8):4223-32.

Cannabinoids selectively inhibit proliferation and induce death of cultured human glioblastoma multiforme cells.
McAllister SD, Chan C, Taft RJ, Luu T, Abood ME, et al.
Journal of neuro-oncology. 2005; 74(1):31-40.

Time-lapse analysis of potential cellular responsiveness to Johrei, a Japanese healing technique.
Taft R, Moore D, Yount G.
BMC complementary and alternative medicine. 2005; 5:2.

Berberine sensitizes human glioma cells, but not normal glial cells, to ionizing radiation in vitro.
Yount G, Qian Y, Moore D, Basila D, West J, et al.
Journal of experimental therapeutics & oncology. 2004; 4(2):137-43.

Biofield perception: a series of pilot studies with cultured human cells.
Yount G, Smith S, Avanozian V, West J, Moore D, et al.
Journal of alternative and complementary medicine (New York, N.Y.). 2004; 10(3):463-7.

Possible influence of infrasound on glioma cell response to chemotherapy: a pilot study.
Yount G, Taft R, West J, Moore D.
Journal of alternative and complementary medicine (New York, N.Y.). 2004; 10(2):247-50.

In vitro test of external Qigong.
Yount G, Solfvin J, Moore D, Schlitz M, Reading M, et al.
BMC complementary and alternative medicine. 2004; 4:5.

Effects of healing intention on cultured cells and truly random events.
Radin D, Yount G.
Journal of alternative and complementary medicine (New York, N.Y.). 2004; 10(1):103-12.

Distant healing intention: definitions and evolving guidelines for laboratory studies.
Schlitz M, Radin D, Malle BF, Schmidt S, Utts J, et al.
Alternative therapies in health and medicine. 2003; 9(3 Suppl):A31-43.

Bioenergy definitions and research guidelines.
Hintz KJ, Yount GL, Kadar I, Schwartz G, Hammerschlag R, et al.
Alternative therapies in health and medicine. 2003; 9(3 Suppl):A13-30.

Transcriptional activation of TRADD mediates p53-independent radiation-induced apoptosis of glioma cells.
Yount GL, Afshar G, Ries S, Korn M, Shalev N, et al.
Oncogene. 2001; 20(22):2826-35.

Fas (APO-1/CD95) signaling pathway is intact in radioresistant human glioma cells.
Yount GL, Levine KS, Kuriyama H, Haas-Kogan DA, Israel MA.
Cancer research. 1999; 59(6):1362-5.

p53 function influences the effect of fractionated radiotherapy on glioblastoma tumors.
Haas-Kogan DA, Kogan SS, Yount G, Hsu J, Haas M, et al.
International journal of radiation oncology, biology, physics. 1999; 43(2):399-403.

Protein kinase B (PKB/Akt) activity is elevated in glioblastoma cells due to mutation of the tumor suppressor PTEN/MMAC.
Haas-Kogan D, Shalev N, Wong M, Mills G, Yount G, et al.
Current biology : CB. 1998; 8(21):1195-8.

Ionizing radiation inhibits chemotherapy-induced apoptosis in cultured glioma cells: implications for combined modality therapy.
Yount GL, Haas-Kogan DA, Levine KS, Aldape KD, Israel MA.
Cancer research. 1998; 58(17):3819-25.

p53-dependent G1 arrest and p53-independent apoptosis influence the radiobiologic response of glioblastoma.
Haas-Kogan DA, Yount G, Haas M, Levi D, Kogan SS, et al.
International journal of radiation oncology, biology, physics. 1996; 36(1):95-103.

Cell cycle synchrony unmasks the influence of p53 function on radiosensitivity of human glioblastoma cells.
Yount GL, Haas-Kogan DA, Vidair CA, Haas M, Dewey WC, et al.
Cancer research. 1996; 56(3):500-6.

Pentylenetetrazole-induced seizures stimulate transcription of early and late response genes.
Yount GL, Ponsalle P, White JD.
Brain research. Molecular brain research. 1994; 21(3-4):219-24.

D1 and D2 dopamine receptor turnover and D2 messenger RNA levels in the neuroleptic-responsive and the neuroleptic nonresponsive lines of mice.
Qian Y, Hitzemann B, Yount GL, White JD, Hitzemann R.
The Journal of pharmacology and experimental therapeutics. 1993; 267(3):1582-90.

Limbic seizures increase cyclophilin mRNA levels in rat hippocampus.
Yount GL, Gall CM, White JD.
Brain research. Molecular brain research. 1992; 14(1-2):139-42.

Biphasic response of hippocampal dynorphin expression following recurrent limbic seizure.
White JD, Yount GL, Lauterborn JC, Krause JE, Gall CM.
Molecular and cellular neurosciences. 1992; 3(1):79-87.